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The pathological characteristics of ALI involve extensive inflammation resulting in harm to alveolar epithelial cells (AECs), breakdown of the pulmonary vascular endothelial barrier and subsequent accumulation of protein-rich inflammatory edema fluid within the alveolar spaces, ultimately causing diffuse alveolar damage (6)

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Coexpressed catalase protects chimeric antigen receptorredirected T cells as well as bystander cells from oxidative stressinduced loss of antitumor activity

Glutamine has been considered an immunomodulatory amino acid in several disease states, yet the mechanisms underlying the therapeutic effects of glutamine supplementation in critical illness remain poorly understood