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Description
The biosynthesis of glutathione (GSH) and the normal function of GPX4 are central to the control of ferroptosis, and the inhibition of GPX4 can increase cellular ferroptosis sensitivity [6, 7]
No animals were excluded from the analysis unless there was clear evidence of technical failure
We also detected a loss of the neuronal cell marker NeuN and an activation of cleaved caspase-3 (Fig
The Genentech Growth Study Group
AD is a chronic neurodegenerative disorder representing the most prevalent form of dementia in modern society
