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Experimental studies consistently demonstrate that magnesium deficiency enhances neutrophil activation and reactive oxygen species (ROS) production while suppressing antioxidant defense systems, thereby promoting lipid peroxidation and oxidative hepatocellular injury ( + redox balance, impair fatty acid -oxidation, and reduce ATP synthesis, collectively compromising hepatocellular energy homeostasis ( Hypomagnesemia disrupts cellular calciummagnesium homeostasis, often leading to relative calcium overload

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Adipose tissue macrophages induce hepatic neutrophil recruitment and macrophage accumulation in mice