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c-Met / proliferation concern is real and unresolved: c-Met is the product of the MET proto-oncogene, and aberrant HGF/c-Met signaling drives several cancers

This mechanism positions GHK as a regulator of fibrosis progression and resolution rather than a direct inhibitor of myofibroblast differentiation markers

Blood DNA methylation profiling identifies cathepsin Z dysregulation in pulmonary arterial hypertension

Get baseline bloodwork Know your starting hormone levels so you can track changes

The thyroid scan was within normal limits
