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This mitochondrial injury promotes the release of apoptotic mediators like cytochrome c and caspases, initiating programmed cell death

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Retinoids work by binding to retinoic acid receptors in the nucleus, directly altering gene transcription

For acute myocardial infarction, hospitals that were not surveyed or accredited had lower performance on care related to that condition, as well as higher mortality rates, than those that were accredited [13]