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To elucidate the role of the AGEs-RAGE pathway in diabetic kidney disease and systematically review therapeutic strategies targeting this pathway.

In this study, potential molecular targets of Q and Q3OG were first identified using the Swiss Target Prediction platform and pathogenic targets of AD were identified using the DisGeNET database

Nevertheless, the precise molecular mechanisms underlying GST-induced drug resistance remain incompletely understood

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Importantly, overexpression of keap1a or knockdown of hmox1a partially rescued the blood clot formation, suggesting that the disrupted Keap1-Nrf2 signaling is responsible for the venous thrombosis-like phenotypes in smarca5 mutants