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83 NSCs have been shown to be an effective neural repair strategy in preclinical models of injury owing to their inherent ability to provide neuroprotection, decrease immunoreactivity, and secrete neurotrophic factors, such as glial-derived neurotrophic factor and brain-derived neurotrophic factor

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Post-translational redox control of APR (particularly APR1) marks it out as a significant control point in response to glutathione oxidation (Leustek, 2002)