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[2] Second, in cultured dermal microvascular and fibroblast models, GHK-Cu exposure has been associated with increased vascular endothelial growth factor expression, a change that could enhance perifollicular microcirculation

This mechanism is completely distinct from growth hormones receptor-mediated actions and explains how AOD-9604 can provide fat metabolism benefits without systemic hormonal effects

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It also modulates nitric oxide pathways and growth factor expression, both of which play critical roles in how your body heals from injury, surgery, or chronic inflammation
