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Lewis base catalysis of bromo- and iodolactonization, and cycloetherification

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Mechanisms of Disease: metabolic effects of growth hormone and insulin-like growth factor 1

173 observed that, in the context of PTEN inactivation, hyperactivated AKT is associated with phosphorylation of 4E-BP1, increased activity of complexes I, III and IV and augmented oxygen consumption

Additionally, abnormalities in fatty acid -oxidation (FAO) and amino acid metabolism can lead to the buildup of lipids and uremic toxins in the kidney, which can contribute to renal cell damage and apoptosis [4, 8]