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Description
Application: For use in controlled laboratory and experimental settings
A shift in mitochondrial substrates and metabolic inflexibility causes DNA mutations, oxidative stress, and apoptosis in PTPMT1 knockout cells
However, the FDA has signaled increasing scrutiny of whether these modifications truly constitute clinical differentiation or are simply cosmetic changes designed to circumvent the ban on copying commercially available drugs
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The median and maximum lifespans observed of control and treated populations for these eight replicates are shown in Supplementary file 4
