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Since vitamin B12 supports red blood cell production (via ODS), it is conceivable that when a vitamin B12 deficiency is treated with high dosage supplementation, the result may be the overproduction of red blood cells

Given that NNMT has been reported to be a primary metabolic enzyme[36] and a major clearance pathway for intracellular NA,[15] it is likely that these small molecules are directly and selectively inhibiting the conversion of NA to 1-MNA