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However, under conditions of iron overload, excess Fe 2+ in the LIP undergoes Fenton chemistry with H 2 O 2 , producing excessive ROS, promoting lipid peroxidation, and ultimately triggering ferroptosis (Zhao et al., 2025a)

In a separate analysis of ApoE / mice on an atherogenic diet for 12 weeks, GPx1 mRNA (detected by in situ hybridization) and GPx1 protein (detected by immunostaining) were highly concentrated in macrophage- and smooth muscle cell (SMC)-rich areas of atherosclerotic lesions of ApoE / aortas [80]

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