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In an effort to investigate whether NMN treatment would have an impact on the health of UMNs diseased with TDP-43 pathology, we utilized dissociated motor cortex cultures established from prpTDP-43 A315T -UeGFP mice, in which CSMN are genetically labeled with eGFP expression that is stable and long-lasting, and when in culture retain their CSMN identity and eGFP expression
Simultaneously, IL-6 and IFN- further reinforce the pro-inflammatory state of microglia via the JAK/STAT axis, enabling the sustained expression of inflammatory cytokines even in the absence of persistent exogenous stimuli (175, 177)
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This makes it suitable for people seeking overall antioxidant support and general skin health improvement rather than targeting specific spots
