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Examples include restricting CB 1 R agonists to peripheral receptors, creating specific CB 2 R agonists, inhibition of endocannabinoid metabolism or reuptake and modulating non-CB 1 R/CB 2 R targets of cannabinoids such as TRPV1, PPARs and GPR55 [127]

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Eicosapentaenoic acid actions on adiposity and insulin resistance in control and high-fat-fed rats: role of apoptosis, adiponectin and tumour necrosis factor-

These interactions stabilize ethacrynic acid within the GSTP1 active site, aligning with its known mechanism of action in reducing GSTP1 activity by interfering with its catalytic function