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As a result of the increased interaction of Ang II with its receptor AT-1, and through the nuclear translocation of NF-B [20], Ang II induces the production of pro-inflammatory cytokines, C-reactive protein (CRP), oxidative stress, fibrosis, and vasoconstriction, and it increases the activity of ADAM17 [21], among other harmful effects

The ascension of targeted covalent inhibitors

Warthen JL, Lueckheide MJ

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