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Extensive evidence suggests that defects in the process of selective autophagy of dysfunctional mitochondria contribute to neurodegeneration (Fivenson et al., 2017), as well as alterations in the mitochondrial unfolded protein response (UPR), which is a signal transduction pathway from mitochondria to the nucleus

Metabolites of microbiota response to tryptophan and intestinal mucosal immunity: A therapeutic target to control intestinal inflammation

To validate this cell death, PI staining revealed more cell death in si-P65 and si-EAAT3 cells following RSL3 treatment (Fig