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A new paper out today in the Journal of Biological Chemistry from Kenneth Johnson, a professor of molecular biosciences, describes how he and his team examined the biochemical mechanism that enables an antiviral drug called sofosbuvir to halt replication in the Hepatitis C virus

Quantification of PUFA C=C isomers was also validated using a series of -3 and -6 FA 18:3 isomer mixtures in a similar way, while moderate epoxidation condition was applied to yield high amount of the mono-epoxidized ( Fig S10, Table S2 )

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The difficulty in identifying the virus and in detecting viral RNA and antigens in infected tissues left the impression that HCV replicated poorly in vivo

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