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High levels of GSH support an anti-inflammatory M2 macrophage phenotype, which is essential for tissue repair and resolving inflammation

The effect on astrocytes may happen independently of Nrf2, since cofilin-1, tubulin and collapsin response mediator protein 2 (CRMP2) activity was seen to be mediated by DMF and reduced cytoskeletal remodeling (173)

aeruginosa does not predominate in a healthy airway, and shifts in the mucin glycosylation patterns or accessibility of the glycans to the sensor are suggested as possibilities for the dominance of P

tuberculosis infection also up-regulates ZnT1 expression in human macrophages, 542 which probably facilitates the increase of zinc levels in macrophage phagosomes in conjunction with M