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It is interesting to point out that the same biological effect can be achieved through Igf-1 by activating different pathways in different cells: IIS induces proliferation in embryonic rat cortical progenitors in an Erk-independent manner, while Igf-1 signalling induces proliferation of adult hippocampal progenitors via Erk-dependent cytoplasmic downstream signals

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We begin with the epidemiological, genetic, and environmental underpinnings of vulnerability to ASD, and on this basis, turn our attention to the current scientific discourse regarding the relationship of the immune system with the nervous system and host microbiota in homeostasis/allostasis, neurodevelopment, and psychological and physiological health and disease