neuronal glutathione synthesis in vivo Impaired Neurodegeneration Mathematical model for glutathione dynamics
Description
135 Dihydroartemisinin can significantly inhibit the viability of leukemia cells, causing cell cycle G0/G1 phase arrest and ferroptosis, and the mechanism may be related to inducing autophagy and accelerating ferritin degradation by regulating AMP-activated kinase (AMPK)/mTOR/p70S6k signaling pathway
At this stage, the therapeutic efficacy of Rg1 remains to be established by further clinical validation, although the initial findings are encouraging
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Promoter activity was determined via the luciferase assay system (Promega) according to the manufacturers instructions
doi: 10.4049/jimmunol.1301490 147 RenJ-DWuX-BJiangRHaoD-PLiuY
