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They are vital for functions like learning, memory, and neuronal communication
SELENOS has a role in the removal of stressor-induced misfolded proteins from the ER, preventing the accumulation of these proteins and the subsequent stress response that leads to activation of NF-B, pro-inflammatory cytokine gene transcription and the inflammation cascade (Fig
2015 , CD003793 (2015)
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Nrf2-mediated redox signaling in arsenic carcinogenesis: a review
