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When tau protein becomes highly phosphorylated, it aggregates, inhibiting microtubule function, impairing neuronal axonal transport, and thus leading to neuronal cytotoxicity 84

[48] demonstrated that deacetylation of iNAMPT by the SIRT1 deacetylase enhances eNAMPT secretion and activity in murine adipocytes

The stimulation of the CREB pathway by XESAD suggests the possibility that XEASD may be effective in treating obsessive compulsive disorder, which occurs in a large percentage of patients with TS (Buzan et al., 2000

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