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In our laboratory, we also showed a differential regulation of human DOR (hDOR) on both the inhibition of ACase and the phosphorylation of ERK1/2 in the SK-N-BE cells

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1(A-D), HG exposure slowed wound closure compared to control cells (CTR, SG condition), and this effect was prevented by Epitalon treatment

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Hematoxylin and eosin (HE) staining and Von-Kossa staining were used to confirm the successful establishment of the model
