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Studies have shown that 20 M or a higher dose of VLCFA can induce mitochondrial inner membrane depolarization and permeability transformation, resulting in excessive ROS, which leads to oxidative stress characterized by lipid peroxidation, protein carbonylation, increased SOD activity, decreased CAT activity, and glutathione level, especially on microglia, astrocytes, and adrenocortical cells (73, 7983)

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