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Taken together, these findings suggest that targeting pro-iron death signaling could represent a promising therapeutic approach for the treatment of senescence and related disorders

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For instance, miR-21 promotes CD4 + T cell glycolysis through the PTEN/PI3K/AKT pathway, which increases the cell cycle, promotes CD4 + T cell polarization toward the Th2 phenotype, and releases the fibrogenic cytokine IL-13, which is involved in arsenite-induced hepatic fibrosis [110] (Fig

The exact mechanism by which sulfa-based agents cause acute angle closure glaucoma is unknown