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In this study, oral ALA (600 mg twice daily) was administered for 4 weeks to 12 patients (mean age 52.9 9.9 years)

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In the intrinsic pathway, mitochondrial damage, such as DNA damage or hypoxia, causes release of cytochrome C, which led to the production of apoptosome and therefore to the activation of caspasce-9, caspase-3, and caspase-7 and apoptosis

doi:10.1186/1742-2094-9-168 Reddy RV, et al

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