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This cancer follows a similar high prevalence pattern of chronic hepatitis B virus (HBV) and approximately 80% of HCC cases occur there [67]

In circulation, RGD peptides are shielded by the POEG coating, and HRNMs achieve effective tumor aggregation through passive targeting

These properties may retard the progression of liver injury and may even facilitate the reversal of hepatic fibrosis in patients with NAFLD who are at risk for developing NASH, as oxidative stress has been implicated as one of the key pathways in NASH pathogenesis [3]

To address this gap, several emerging avenues are being explored [110, 230]

When the fusion with the lysosomes takes place, the contents of the autophagosome are degraded by the resident hydrolases