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Thus, several components of the PI3K/Akt/mTOR pathway frequently show mutations and are activated in cancer (61, 62)

However, sub-lethal damage is characterized by the disruption of tight junctions between tubular epithelial cells, detachment of these cells from the basement membrane, and the shedding of apical membrane fragments into the tubular lumen

In the present study, we fused CcSOD1 with a cell-penetrating peptide TAT in order to deliver CcSOD1 protein into cells and explore its therapeutic prospects

In theory, these are all positive things if muscle growth is the desired outcome, but it is unproven (12, 13)

Strazzabosco M