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HMGB1 binds to cell membrane receptors (RAGE, TLR2, TLR4, and TREM-1), activating NLRP3, which assembles apoptosis-associated speckle-like protein (ASC) and recruits caspase-1 for processing (169)
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Marc Dunoyer, the companys rare disease head, in a statement called Ultomiris a transformative treatment option for NMOSD, with the potential to eliminate relapses with a convenient dosing schedule every eight weeks. The approval was backed by data from a Phase III trial that met its primary efficacy endpoint, preventing relapse in all 58 treated patients over a median of 73.5 weeks, compared to 20 relapses in the placebo arm of a trial for AstraZenecas other NMOSD therapy Soliris
[DOI] [PMC free article] [PubMed] [Google Scholar] Hausmann G, Banziger C, Basler K
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