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Description
An improved understanding of the role oxidative stress plays in neural injury in sleep apnea may be developed by integrating information gained examining neural tissue in animal models of sleep apnea with key features of redox biochemistry and clinical sleep apnea studies where extra-neuronal oxidative stress characterizations have been performed
The increase of -H2AX adds to the hypothesis that HnB may be associated with DNA-damaging activity
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doi: 10.1186/ar4090
