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However, healthy individuals with intact B12 metabolism convert cyanocobalamin efficiently

Collectively, organ age gap associations with disease and blood biochemistry demonstrate that aging models derived from organ-specific plasma proteins capture disease-relevant heterogeneity of aging within and across individuals, which is not captured by other aging clocks or clinical markers

In the context of Alzheimer's disease, the buildup of amyloid-beta (A) in cerebral mitochondria disrupts the electron transport chain (ETC), resulting in significant ATP depletion and bioenergetic impairment (196)

Aykac-Toker, G., Bulgurcuoglu, S

Additional research, including studies from our collaborative research groups, has shown that severe reactive astrocytes exhibit the increased production of H 2 O 2 through MAO-B, leading to AD pathology hallmarks such as tauopathy, cognitive decline and neuronal atrophy 57