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226 Moreover, HIF activation promotes cell proliferation and inhibits apoptosis in renal IR-induced AKI, and these effects can be reversed by treatment with the -catenin inhibitor IWR-1-endo, indicating interaction between HIF and the Wnt/-catenin signaling pathway in providing renal protection

doi: 10.1371/journal.pone.0131595

Such regulation would prevent an excessive proinflammatory response, adequate enough to inhibit the growth of tumor cells, where it has been suggested that some macrophage membrane receptors (LR2, TLR4, CR3, MR) could be involved (Zhang et al., 2017b)

This was predominant in the male Gclc L-KO mice and was not rescued with Nrf2 deletion (Fig

Dill MT, Tornillo L, Fritzius T, Terracciano L, Semela D, Bettler B, et al