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DOI:10.1186/1743-7075-9-35 [ Links ] 14

Understanding the exact mechanisms by which homocysteine contributes to neurological disorders and whether it is a causative factor or a consequence is still an active area of research

Modulation of dopamine D1 [239], D2 [240], and GABA [241], receptors by Hg, may also involve Hg-thiol group interaction, although the particular target Cys residues have yet to be identified (Table 1)

Inactivation of GPX4, through depletion of GSH or by using GPX4 inhibitors, ultimately results in overwhelming lipid peroxidation and cell death in various non-erythroid cells (Yang et al., 2014

Please see recent reviews on SOD1 ALS for more details [10]