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ATG and Dox co-treatment was induced by downregulating expression of Bcl-xl and Bcl-2, through non-major (or off-target) drug effects by promoting the translocalization of Bax to mitochondria, thereby destroying mitochondrial integrity [58]

Szeto-Schiller peptide 31 Class: Mitochondrial-targeting tetrapeptide

Moreover, c-Myc loss impairs polyamine synthesis, due to both upstream defects in glutaminolysis and decreased expression of polyamine biosynthesis genes

(2014) demonstrated that Al stress increases localized auxin biosynthesis in the root apex transition zone via the Trp aminotransferase 1 (TAA1)-dependent pathway

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