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However, we have previously observed similar effects with CPT1 and CPT1A inhibition considering inflammatory- and oxidative stress levels in other animal models mimicking central nervous system diseases using female rodents 28,29,30,82

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Although the available evidence suggests that ketone bodies are not a factor in the effect of the ketogenic diet on inhibitory synaptic function, ketone bodies have been found to increase brain synaptosomal GABA content [51], as occurs with the anticonvulsant vigabatrin [52], raising the possibility that ketone bodies might have functional effects similar to those of vigabatrin