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Description
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L-arginine and citrulline supplementation can enhance NO production, potentially reducing the frequency and severity of SLEs [16]
In addition, by binding an acetyl group, l-carnitine can maintain the levels of Acetyl-CoA and coenzyme A, playing its buffering role
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Hypertrophic changes in the myocardium are likely a result of energy deficits due to impaired oxidative phosphorylation, leading to myocardial hypertrophy as the heart attempts to compensate for the loss of efficient energy production [1,2,39]
