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Perhaps the strongest case for a role of nitric oxide in neuronal signaling in the CNS is for long-term depression of synaptic transmission in the cerebellum

LpdA is homologous to Gor and the mutant form of this protein may perform a similar function as Gor in order to reduce disulfide bonds in these suppressors, or may be able to reduce oxidized lipoamide and transfer electrons to ribonucleotide reductase via a novel mechanism