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NOS3 variants that reduce eNOS activity or expression add a compounding layer: hypertension, coronary vasospasm, coronary artery disease, stroke, and preeclampsia are all associated with reduced eNOS function and impaired NO bioavailability

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The HOPS complex together with GTPase RAB7 is critical for tethering autophagosomes with lysosomes followed by fusion of the membrane bilayers 24,43,44,45,46 (schemed in Fig

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[DOI] [PubMed] [Google Scholar] 18.Nayki U., Onk D., Balci G., Nayki C., Onk A., ankaya M., Kafa A.H.T., Kuzucu M