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In some experiments on spontaneously hypertensive rats, it has been found that catalpol treatment upregulates several regulatory proteins during prefrontal cortex development [152]

During the initial meeting, the premise was identified and the overall methodology was defined

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doi: 10.1002/hep.31311 34 DubinkinaVBTyakhtAVOdintsovaVYYaryginKSKovarskyBAPavlenkoAVet al

Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis