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Dehydroascorbic acid is reduced to generate ascorbic acid inside the cell, protecting mitochondria from free radical-induced oxidative damage (Figures 2 and 3)

Molecular and cellular processes, such as myofibroblast/mesenchymal transition, myofibroblast activation and uncontrolled proliferation, endoplasmic reticulum stress, altered expression of growth factors, and oxidative stress, link PF to lung cancer and increase the risk of cancer development by 7%20% ( In addition, lung damage and fibrosis are frequently observed in tuberculosis (Ravimohan et al., 2018)

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This impairs the elimination of lipid peroxides and sensitizes cells to ferroptosis

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