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Furthermore, immunofluorescence staining of neutrophils in liver tissue and the analysis of serum levels of TNF and IL-6 revealed that HK2 overexpression markedly attenuated the inflammatory response induced by I/R treatment (Supplementary Fig

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Moreover, the expression level of the main subunit of CII (succinate dehydrogenase A, Sdha) was decreased in Gclc -deficient FoB (Fig
