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Taken together, these findings highlight how lipid dysregulation may be a unifying mechanism linking high UPF intake to pathophysiological changes underpinning anxiety
S., Juillerat-Jeanneret, L
This combination is extensively studied in models investigating muscle recovery, metabolic function, and cellular regeneration
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This study revealed that stimulating GPR55 by its endogenous ligand L- lysophosphatidylinositol (LPI) stimulates cell migration and invasion in an MDA-MB-231 cell line
