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Specifically, NAD+ treatment inhibited the aberrant serine-specific phosphorylation of key BER enzymes, AP endonuclease (APE) and DNA polymerase- (-pol), which are typically inactivated during ischemic conditions

doi: 10.3109/10409239509083491

Doxorubicin (DOX), a widely used anticancer drug, exerts its deleterious effects on cellular homeostasis by downregulating the expression of GPx4 and inducing excessive lipid peroxidation within mitochondria

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