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In cases of chronically sustained Type II GPCR activation, -Arrestins can induce the internalization of the GPCR through clathrin-mediated endocytosis, insulating the receptor from extracellular signals

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However, protein degradation is enhanced when cells are studied in vitro

cAMP response element binding-protein, a transcription factor phosphorylated by PKA that binds to DNA and initiates gene transcription