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Given these known effects of small Mafs and c-Maf, we speculated that the induction in Mafs and displacement of Nrf2 from nuclear binding to ARE during cholestasis might have caused the fall in the expression of GSH synthetic enzymes

Isoenzyme- and allozyme-specific inhibitors: 2,2-dihydroxybenzophenones and their carbonyl N-analogues that discriminate between human glutathione transferase A1-1 and P1-1 allozymes

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Glutathione is already synthesised in the body, and supplementation is the support of the already existing pathways instead of presenting a stranger

Functionalized nanoparticles for brain targeted BDNF gene therapy to rescue Alzheimers disease pathology in transgenic mouse model