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Studies examining mitochondrial enzyme activities reveal that age-related decrements in tricarboxylic acid (TCA) cycle enzymes and electron transport chain complexes are reversed by carnitine supplementation

In vitro post-oxidative stress glaucoma was controlled by carnitine through reducing pathologic optic-disk excavation, typical cell stress markers such as caspase 3 and ubiquitin, inducible nitric oxide synthase, and glial fibrillary acidic protein expression [70]

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Cutting down the fat intake and increased levels of MOTS-c may lower the diet-induced metabolic dysfunction and associated inflammation