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MartnezRevelles S., GarcaRedondo A

2016 doi: 10.3390/nu8010044

Metabolic reprogramming driven by oncogenes allows cancer cells to maintain deregulated cell proliferation, withstand metabolic challenges that are associated with diminished oxygen and nutrients, maintain a dedifferentiated state through alterations in global patterns of gene expression, and corrupt the surrounding microenvironment to actively facilitate tumor growth and dissemination 16,17,18

Specifically, we examined changes in the tissue concentrations of both the reduced (GSH) and oxidized (GSSG) forms of glutathione, in addition to the ratio of GSH to GSSG

A: Long-term studies on GHK-Cu are limited, but available evidence suggests a favorable safety profile with minimal risks when used appropriately