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Furthermore, AKT may directly activate the nicotinamide adenine nucleotide kinase (NADK) responsible for the phosphorylation of NADH forming NADPH (Figure 2
More importantly, as a natural chemical molecule, the targets of TMAO in vivo are not fully elucidated
Amphipathic lipid metabolites and their relation to arrhythmogenesis in the ischemic heart
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