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Furthermore, this result suggests that non-tumorigenic tissues or cells in the tumour microenvironment with high GGT activity can drive tumour growth and progression by catabolizing GSH and supplying amino acids in a paracrine (or endocrine) manner

This rescue by GSH was not observed after deprivation of other amino acids (serine, glycine, glutamine or glutamate) or after inhibition of amino acid synthesis (via the glutaminase 1 inhibitor CB-839) (Extended Data Fig