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Increased LTCC and aberrant NMDA-receptor function impair Ca 2+ homeostasis, and is further enhanced by a decrease in activity of Ca 2+ buffering proteins, driving an (4) altered gene expression which results in Ca 2+ dysregulation, leading to cognitive decline and neurodegeneration

Chen C, et al

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Opoku-Temeng C, Kobayashi SD, DeLeo FR

The glutathione S -conjugate, cysteinylglycine S -conjugate, cysteine S -conjugate, and N -acetylcysteine S -conjugate (mercapturate) derived from halogenated alkenes are all nephrotoxic/hepatotoxic and in many cases nephrocarcinogenic/hepatocarcinogenic in experimental animals (e.g., Anders, 2004, Anders, 2008, Anders and Dekant, 1994, Anders et al., 1988, Boogard et al., 1989, Dekant, 2001, Dekant, 2003, Koob and Dekant, 1991, McGoldrick et al., 2003, Lash et al., 2014, Yaqoob et al., 2014, Cristofori et al., 2015)